Reviewers you may be interested in

Vivek Kumar

Total points: 152

Country: United States

Keywords: Mental disorders; Mental Illness; Psychiatric disorders; Image analysis; Brain transparency; Postmortem Human brain; Feeding Circuitry; Stress; Anxiety; Noise; Prenatal auditory stimulus; Light sheet microscopy; Fluorescence in situ hybridization; Tissue clearing; iDISCO

Vladimir Achikyan

Total points: 105

Country: Russian Federation

Keywords: Immunology, Rheumatology, inflammatory bowel disease, colitis, musculoskeletal disorders, pain, sleep disorders, HCV, liver disease, HIV, AIDS, Oncology, breast cancer, lung cancer, melanoma, Oncourology, clinical trials, clinical studies, real world data, real world evidence

Xin Xie

Total points: 97

Country: China

Keywords: Oncology, antibiotics, biochemistry, molecular biology, natural products, pharmacology, medicinal chemistry, structure-activity relationship, herbal, traditional medicine, alternative medicine

Nezih Oktar

Total points: 78

Country: Turkey

Keywords: Neurooncology Neurosurgery,Vascular Neurosurgery,Neurooncology,Neurosurgery,Glioblastoma,Mitochondria

Mikhail Matveyenka

Total points: 74

Country: United States

Keywords: PD, AD, Amyloids, Oncology, Immunotherapy, Cells Therapy, Immunology

xinna li

Total points: 72

Country: United States

Keywords: I have strong research experience in molecular biology and biochemistry. In 2012, I joined the group of Richard Miller to study the mechanism of aging using longevity mouse models. My work has provided encouraging evidence that ARIs (Aging Rate Indicators) may be altered by genes, CR diet, and drugs in many tissues of many varieties of slow aging mice. The slow aging is attributed to a multitude of causes including mutations (Ames, Snell, GHR-KO, or PAPPA-KO) and CR diet, mice treated with rapamycin (Rapa), acarbose (Aca), 17aE2, and canagliflozin (Cana).These genes and interventions lead to many physiological changes such as 1) an increase in uncoupling protein UCP1 in brown and white adipose tissue (WAT); 2) a change in fat-associated macrophage subsets that leads to diminished production of inflammatory cytokines; 3) an increase in muscle fibronectin type III domain containing 5 (FNDC5) and its cleavage product, the myokine irisin, thought to cause changes in fat cell differentiation; 4) elevated production of hepatic GPLD1 and plasma GPLD1; 5) elevation of hippocampal BDNF and DCX, used as indices of neurogenesis.