Reviewers you may be interested in

Dr. Eknath Ahire

Total points: 168

Country: India

Keywords: Quality control, Formulation, Food, Nutraceuticals, Drug delivery,Medicines,Fromulation Deveopment,Therapy,Cancer,Drug resistance

Amir Miri

Total points: 129

Country: Iran (the Islamic Republic of)

Keywords: Antibiotics, antibiotic resistance, nanoparticles, nanomedicine, active targeting, passive targeting, surface functionalization, controlled release, microparticles, probiotics, gut microbiota, gastric microbiota, infectious diseases, microbiome, Helicobacter pylori, drug delivery systems, Alternatives to antibiotics.

Vladimir Achikyan

Total points: 105

Country: Russian Federation

Keywords: Immunology, Rheumatology, inflammatory bowel disease, colitis, musculoskeletal disorders, pain, sleep disorders, HCV, liver disease, HIV, AIDS, Oncology, breast cancer, lung cancer, melanoma, Oncourology, clinical trials, clinical studies, real world data, real world evidence

Mehmet Micoogullari

Total points: 84

Country: Turkey

Keywords: Musculoskeletal Rehabilitation, Sports Health, Sporty Performance, Temporomandibular Joint, Spine health

Xiaohe Jin

Total points: 76

Country: United States

Keywords: Biosensors; Photosynthesis; Synthetic Biology; Nano(bio)materials; Self-assembly of biomolecular materials; Gene/Drug delivery systems; DNA Nanostructure Scaffolds; Hydrogels; Nanobiotechnology; Cell Imaging.

xinna li

Total points: 72

Country: United States

Keywords: I have strong research experience in molecular biology and biochemistry. In 2012, I joined the group of Richard Miller to study the mechanism of aging using longevity mouse models. My work has provided encouraging evidence that ARIs (Aging Rate Indicators) may be altered by genes, CR diet, and drugs in many tissues of many varieties of slow aging mice. The slow aging is attributed to a multitude of causes including mutations (Ames, Snell, GHR-KO, or PAPPA-KO) and CR diet, mice treated with rapamycin (Rapa), acarbose (Aca), 17aE2, and canagliflozin (Cana).These genes and interventions lead to many physiological changes such as 1) an increase in uncoupling protein UCP1 in brown and white adipose tissue (WAT); 2) a change in fat-associated macrophage subsets that leads to diminished production of inflammatory cytokines; 3) an increase in muscle fibronectin type III domain containing 5 (FNDC5) and its cleavage product, the myokine irisin, thought to cause changes in fat cell differentiation; 4) elevated production of hepatic GPLD1 and plasma GPLD1; 5) elevation of hippocampal BDNF and DCX, used as indices of neurogenesis.