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Adrià Arboix

[Available for Review]

H-index: 58

Manuscripts in Review: 0

Reviewed Articles: 6

Invitation Reply Cycle: 24 h

Total Points: 79

Manuscript Review Cycle: 7 days

Country: Spain

Institution: --

Section: Medicine, General & Internal

Keywords: acute stroke, cerebrovascular diseases, TIA, vascular dementia

Review journals:

Exploration of Neuroscience (4)    Exploration of Neuroprotective Therapy (1)    Exploration of Cardiology (1)   

 A dash (--) means information awaiting reviewer update.

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xinna li

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Reviewed Articles: 7

Keywords: I have strong research experience in molecular biology and biochemistry. In 2012, I joined the group of Richard Miller to study the mechanism of aging using longevity mouse models. My work has provided encouraging evidence that ARIs (Aging Rate Indicators) may be altered by genes, CR diet, and drugs in many tissues of many varieties of slow aging mice. The slow aging is attributed to a multitude of causes including mutations (Ames, Snell, GHR-KO, or PAPPA-KO) and CR diet, mice treated with rapamycin (Rapa), acarbose (Aca), 17aE2, and canagliflozin (Cana).These genes and interventions lead to many physiological changes such as 1) an increase in uncoupling protein UCP1 in brown and white adipose tissue (WAT); 2) a change in fat-associated macrophage subsets that leads to diminished production of inflammatory cytokines; 3) an increase in muscle fibronectin type III domain containing 5 (FNDC5) and its cleavage product, the myokine irisin, thought to cause changes in fat cell differentiation; 4) elevated production of hepatic GPLD1 and plasma GPLD1; 5) elevation of hippocampal BDNF and DCX, used as indices of neurogenesis.
Sai Shilpa Kommaraju

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Paolo BONGIOANNI

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